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Actual for You - Pain Anatomy 101: How Pain Works, And How Your Subconscious May Be Able to Short-Circuit It
A new ebook, called GetGoogleAdsFree, claims that a New York business man got more than one million dollars worth of Google's PPC ads free of cost. It went on to say that he did not get them illegally and that Google in fact did not mind.To be completely honest with you, you cannot get free Google adwords PPC ads. Google makes a lot of money through Adwords and its sister program, Adsense. What would happen if every one suddenly had a way to get these PPC ads for free?So what are they talking about?The author of the ebook, GetGoogleAdsFree, knows that you cannot get free Google Adwords PPC ads. However, they mislead audiences into thinking they can since Google's PPC ads have become more competitive over the years and cost per click has risen. People want to get lower costs and would want a way out. Getting free Google Adwords PPC ads would be that way out.The system however is far from free. If anyone is going to use this system then they would have to be advanced users of
Stage 2 (Spinal Cord to Thalamus): The Source of Major Pain
When you have chronic pain, a series of events can occur in Stage 2 to radically increase the pain signal:
Ion Channels. Ion channels may be allowing an unusually large amount of pain chemicals to move into the Stage 2 nerve cells (see Stage 1 above).
Interneuron Windup. Normally C-fibers and A-beta fibers don't talk to each other very much. But in chronic pain they may--the repetitive firing of C-fibers can super-sensitize the big A-beta fibers. In turn, the A-betas trigger the first Level 2 neurons they find, the interneurons, to fire uncontrollably--even long after the stimulus is shut off. This is called interneuron windup and greatly increases the pain signal.
NMDA Activation. Interneurons connect Level 1 to Level 2 neurons. When they send a strong pain signal to the main Level 2 nerve cells, glutamate is released and a powerful protein called NMDA is activated. This ratchets pain up to a new level, and makes possible the next two events, which t
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Science doesn't fully understand how pain signals are generated in the periphery of the body and transmitted to the brain. But it knows the major mechanisms well enough to be able to identify a number of weak points where the signals could be reduced or interrupted. These can be referred to as pain leverage points.
Here's a recap of our understanding of the process based on publicly available information. It's a great oversimplification, and may not occur in all cases. But it gives you a general idea of what's happening.
This mechanism seems to be operating in back pain or pain in other extremities, arthritis pain, fibromyalgia pain, or neuropathic pain (nerve pain).
Please note that we are not medical professionals and do not provide medical or health care advice. For medical advice, please see your physician.
Note also that the material in this article is provided to help you understand the details of the pain process and how you can attempt to relieve pain by using your subconscious. But this information is optional you don't need to read or understand any of it before attempting to use your subconscious to ease pain.
A Three-Stage Process
In general, pain signals move through three steps. Each of these steps is performed by a different type of nerve cell, or neuron:
Stage 1: The pain signal begins in the periphery of the body--the skin, the lower back, anyplace that receives a painful stimulus. In Stage 1, Level 1 neurons pick up the signal and send it to the spinal cord for processing.
Stage 2: In Stage 2, Level 2 neurons in the spinal cord make modifications to the signal. Here the signal can be radically increased. The Level 2 neurons send the modified signal to the thalamus, the pain processing center in the brain, but at this point you don't yet feel any pain.
Stage 3: In Stage 3, Level 3 neurons in the brain move the signal from the thalamus to the cortex, where you finally experience it as pain.
Neurons are extremely long. A single Level 1 cell can stretch from your big toe to your spine. A single Level 2 cell can reach from your spine all the way up to the thalamus in your brain. And a single Level 3 cell in the brain can reach from the thalamus to the cortex.
The Role of Visualization Statements
We strive to use visualization statements to help the subconscious reduce the pain signal at 12 different leverage points along its 3-stage pathway.
Visualization statements represent the specific language that your subconscious wants you to read back to it to help ease your pain. Theyre simple and are targeted directly at the main factors that could bring you relief.
You can obtain these statements by learning how to communicate directly with your own subconscious mind. The process is straightforward and can be done at home by working with a facilitator over the telephone. You you need no special skills and no previous experience in working with the subconscious.
Stage 1 (Periphery to Spinal Cord): It's All About Inflammation
The body's first response to painful stimulation is usually inflammation. Pain from this source can occur for the following reasons:
Prostaglandins. Most often inflammation is generated by hormone-like prostaglandins that are released at the site of injury or other stimulus. These chemicals inflame the two main kinds of Level 1 neurons: (1) C-fibers, thin bundles of neurons that need strong stimulation to fire, and don't identify the location of the pain very well. (2) A-beta fibers, thick bundles of neurons that fire from either light or strong stimulation, and pinpoint the exact location of the pain.
Substance P. Normally pain signals are carried towards the brain, but in chronic pain the signals can get reversed and be carried back down into the body. If this applies to you, your body could be releasing a protein called substance P, powerfully inflaming nerve endings in some locations.
Ion Channels. The pain signals from the above two sources of inflammation can be made worse if ion channels have forced openings in the nerve cell walls. These openings allow an abnormal increase of pain chemicals to move into the neuron. Ion channels are potentially a problem in all three stages on the pain signal pathway.
Prostaglandins, substance P, and ion channels can all be addressed using visualization statements. It's possible that this may help to reduce inflammation in Stage 1.
Stage 2 (Spinal Cord to Thalamus): The Source of Major Pain
When you have chronic pain, a series of events can occur in Stage 2 to radically increase the pain signal:
Ion Channels. Ion channels may be allowing an unusually large amount of pain chemicals to move into the Stage 2 nerve cells (see Stage 1 above).
Interneuron Windup. Normally C-fibers and A-beta fibers don't talk to each other very much. But in chronic pain they may--the repetitive firing of C-fibers can super-sensitize the big A-beta fibers. In turn, the A-betas trigger the first Level 2 neurons they find, the interneurons, to fire uncontrollably--even long after the stimulus is shut off. This is called interneuron windup and greatly increases the pain signal.
NMDA Activation. Interneurons connect Level 1 to Level 2 neurons. When they send a strong pain signal to the main Level 2 nerve cells, glutamate is released and a powerful protein called NMDA is activated. This ratchets pain up to a new level, and makes possible the next two events, which to
We all make mistakes when selling our product or service. Here are the most common mistakes people make. I have to admit I have made many of mistakes listed in this article even though I have been teaching this stuff for almost a decade. I hope you can learn from them.1. Allowing a prospect to lead the sales process. The best way to control the sales interaction is to ask questions. This is also the best way of learning whether or not your product or service meets the needs of your prospect. Quality questions that uncover specific issues, problems, or corporate objectives are essential in helping you establish yourself as an expert.2. Not completing pre-meeting research. After several weeks of voice mail I finally connected with my prospect and scheduled a meeting. Unfortunately, I entered the meeting without first researching the company. Instead of presenting a solution to an existing problem, I spent the entire meeting learning fundamental information, which to senior executives, is a
A Three-Stage Process
In general, pain signals move through three steps. Each of these steps is performed by a different type of nerve cell, or neuron:
Stage 1: The pain signal begins in the periphery of the body--the skin, the lower back, anyplace that receives a painful stimulus. In Stage 1, Level 1 neurons pick up the signal and send it to the spinal cord for processing.
Stage 2: In Stage 2, Level 2 neurons in the spinal cord make modifications to the signal. Here the signal can be radically increased. The Level 2 neurons send the modified signal to the thalamus, the pain processing center in the brain, but at this point you don't yet feel any pain.
Stage 3: In Stage 3, Level 3 neurons in the brain move the signal from the thalamus to the cortex, where you finally experience it as pain.
Neurons are extremely long. A single Level 1 cell can stretch from your big toe to your spine. A single Level 2 cell can reach from your spine all the way up to the thalamus in your brain. And a single Level 3 cell in the brain can reach from the thalamus to the cortex.
The Role of Visualization Statements
We strive to use visualization statements to help the subconscious reduce the pain signal at 12 different leverage points along its 3-stage pathway.
Visualization statements represent the specific language that your subconscious wants you to read back to it to help ease your pain. Theyre simple and are targeted directly at the main factors that could bring you relief.
You can obtain these statements by learning how to communicate directly with your own subconscious mind. The process is straightforward and can be done at home by working with a facilitator over the telephone. You you need no special skills and no previous experience in working with the subconscious.
Stage 1 (Periphery to Spinal Cord): It's All About Inflammation
The body's first response to painful stimulation is usually inflammation. Pain from this source can occur for the following reasons:
Prostaglandins. Most often inflammation is generated by hormone-like prostaglandins that are released at the site of injury or other stimulus. These chemicals inflame the two main kinds of Level 1 neurons: (1) C-fibers, thin bundles of neurons that need strong stimulation to fire, and don't identify the location of the pain very well. (2) A-beta fibers, thick bundles of neurons that fire from either light or strong stimulation, and pinpoint the exact location of the pain.
Substance P. Normally pain signals are carried towards the brain, but in chronic pain the signals can get reversed and be carried back down into the body. If this applies to you, your body could be releasing a protein called substance P, powerfully inflaming nerve endings in some locations.
Ion Channels. The pain signals from the above two sources of inflammation can be made worse if ion channels have forced openings in the nerve cell walls. These openings allow an abnormal increase of pain chemicals to move into the neuron. Ion channels are potentially a problem in all three stages on the pain signal pathway.
Prostaglandins, substance P, and ion channels can all be addressed using visualization statements. It's possible that this may help to reduce inflammation in Stage 1.
Stage 2 (Spinal Cord to Thalamus): The Source of Major Pain
When you have chronic pain, a series of events can occur in Stage 2 to radically increase the pain signal:
Ion Channels. Ion channels may be allowing an unusually large amount of pain chemicals to move into the Stage 2 nerve cells (see Stage 1 above).
Interneuron Windup. Normally C-fibers and A-beta fibers don't talk to each other very much. But in chronic pain they may--the repetitive firing of C-fibers can super-sensitize the big A-beta fibers. In turn, the A-betas trigger the first Level 2 neurons they find, the interneurons, to fire uncontrollably--even long after the stimulus is shut off. This is called interneuron windup and greatly increases the pain signal.
NMDA Activation. Interneurons connect Level 1 to Level 2 neurons. When they send a strong pain signal to the main Level 2 nerve cells, glutamate is released and a powerful protein called NMDA is activated. This ratchets pain up to a new level, and makes possible the next two events, which t
Unfortunately, very few people have "perfect credit" but having made some mistakes in the past does not mean there is not a product for you. No credit is just that. This means that the person has no information pertaining to their payment history. The good thing is that there are other things that can be taken into consideration to show you have the ability and willingness to pay your debts. One positive thing is a history of rent payments. Another thing to show is a cell phone or land line telephone bill. Utility bills are another way to show a history of paying bills. Simply having no file does not bar a person from obtaining home financing. There is no such thing as having no credit history. There is always something available to show a history of payment.Slow credit is another possibility and is defined by someone who does pay there bills but has some delinquency payments, just paying a little slower than when they are due. Late payments affect your credit based on the severity. Reporting ag
The Role of Visualization Statements
We strive to use visualization statements to help the subconscious reduce the pain signal at 12 different leverage points along its 3-stage pathway.
Visualization statements represent the specific language that your subconscious wants you to read back to it to help ease your pain. Theyre simple and are targeted directly at the main factors that could bring you relief.
You can obtain these statements by learning how to communicate directly with your own subconscious mind. The process is straightforward and can be done at home by working with a facilitator over the telephone. You you need no special skills and no previous experience in working with the subconscious.
Stage 1 (Periphery to Spinal Cord): It's All About Inflammation
The body's first response to painful stimulation is usually inflammation. Pain from this source can occur for the following reasons:
Prostaglandins. Most often inflammation is generated by hormone-like prostaglandins that are released at the site of injury or other stimulus. These chemicals inflame the two main kinds of Level 1 neurons: (1) C-fibers, thin bundles of neurons that need strong stimulation to fire, and don't identify the location of the pain very well. (2) A-beta fibers, thick bundles of neurons that fire from either light or strong stimulation, and pinpoint the exact location of the pain.
Substance P. Normally pain signals are carried towards the brain, but in chronic pain the signals can get reversed and be carried back down into the body. If this applies to you, your body could be releasing a protein called substance P, powerfully inflaming nerve endings in some locations.
Ion Channels. The pain signals from the above two sources of inflammation can be made worse if ion channels have forced openings in the nerve cell walls. These openings allow an abnormal increase of pain chemicals to move into the neuron. Ion channels are potentially a problem in all three stages on the pain signal pathway.
Prostaglandins, substance P, and ion channels can all be addressed using visualization statements. It's possible that this may help to reduce inflammation in Stage 1.
Stage 2 (Spinal Cord to Thalamus): The Source of Major Pain
When you have chronic pain, a series of events can occur in Stage 2 to radically increase the pain signal:
Ion Channels. Ion channels may be allowing an unusually large amount of pain chemicals to move into the Stage 2 nerve cells (see Stage 1 above).
Interneuron Windup. Normally C-fibers and A-beta fibers don't talk to each other very much. But in chronic pain they may--the repetitive firing of C-fibers can super-sensitize the big A-beta fibers. In turn, the A-betas trigger the first Level 2 neurons they find, the interneurons, to fire uncontrollably--even long after the stimulus is shut off. This is called interneuron windup and greatly increases the pain signal.
NMDA Activation. Interneurons connect Level 1 to Level 2 neurons. When they send a strong pain signal to the main Level 2 nerve cells, glutamate is released and a powerful protein called NMDA is activated. This ratchets pain up to a new level, and makes possible the next two events, which t
Implementing an effective employee motivation program is something that most managers and human resource professionals struggle with on some level. The question always seems to come back to what methods and approaches one should use to get the most bang for their buck.There have been many, many studies on the subject over the years, ranging from the Hawthorne Studies that were conducted by Elton Mayo in the 1920's and 1930's, through the current day. They all reach the same basic conclusions. Some take 150 pages and others --like Frederick Herzberg's excellent One More Time: How Do You Motivate Employees article-- cover the bases in just a few pages.The gist of all of the data is that people are competitive creatures. They would like a certain equity in the workplace, but at the same time they crave personal recognition for doing their job well.Managers often view the Equity part of the equation as a Pay Equity situation. When in fact it really isn't. Unless there is a wide d
Substance P. Normally pain signals are carried towards the brain, but in chronic pain the signals can get reversed and be carried back down into the body. If this applies to you, your body could be releasing a protein called substance P, powerfully inflaming nerve endings in some locations.
Ion Channels. The pain signals from the above two sources of inflammation can be made worse if ion channels have forced openings in the nerve cell walls. These openings allow an abnormal increase of pain chemicals to move into the neuron. Ion channels are potentially a problem in all three stages on the pain signal pathway.
Prostaglandins, substance P, and ion channels can all be addressed using visualization statements. It's possible that this may help to reduce inflammation in Stage 1.
Stage 2 (Spinal Cord to Thalamus): The Source of Major Pain
When you have chronic pain, a series of events can occur in Stage 2 to radically increase the pain signal:
Ion Channels. Ion channels may be allowing an unusually large amount of pain chemicals to move into the Stage 2 nerve cells (see Stage 1 above).
Interneuron Windup. Normally C-fibers and A-beta fibers don't talk to each other very much. But in chronic pain they may--the repetitive firing of C-fibers can super-sensitize the big A-beta fibers. In turn, the A-betas trigger the first Level 2 neurons they find, the interneurons, to fire uncontrollably--even long after the stimulus is shut off. This is called interneuron windup and greatly increases the pain signal.
NMDA Activation. Interneurons connect Level 1 to Level 2 neurons. When they send a strong pain signal to the main Level 2 nerve cells, glutamate is released and a powerful protein called NMDA is activated. This ratchets pain up to a new level, and makes possible the next two events, which t
As most successful internet marketers, authors, webmasters and business owners will agree, freelance writers are heaven-sent!People hire ghostwriters for many reasons. Some people are very busy. When you are running a big business and managing dozens of sites that need fresh content on a regular basis, outsourcing makes perfect sense. In some cases, people do not know a lot about the topic they want to have an e-book written on, and they do not have the time to do the necessary research for writing the e-book. Engaging ghostwriters to write your articles or e-books makes perfect sense if you are building one or more content sites.Ghostwriters or freelance writers give a huge boost to your productivity by allowing you to outsource the tedious but important task of writing your e-books or web content. Established internet marketers with many websites do not have the time to write to write all their content because they have better things to do such as marketing the websites or launching n
Stage 2 (Spinal Cord to Thalamus): The Source of Major Pain
When you have chronic pain, a series of events can occur in Stage 2 to radically increase the pain signal:
Ion Channels. Ion channels may be allowing an unusually large amount of pain chemicals to move into the Stage 2 nerve cells (see Stage 1 above).
Interneuron Windup. Normally C-fibers and A-beta fibers don't talk to each other very much. But in chronic pain they may--the repetitive firing of C-fibers can super-sensitize the big A-beta fibers. In turn, the A-betas trigger the first Level 2 neurons they find, the interneurons, to fire uncontrollably--even long after the stimulus is shut off. This is called interneuron windup and greatly increases the pain signal.
NMDA Activation. Interneurons connect Level 1 to Level 2 neurons. When they send a strong pain signal to the main Level 2 nerve cells, glutamate is released and a powerful protein called NMDA is activated. This ratchets pain up to a new level, and makes possible the next two events, which together can produce devastating pain.
MAPK Activation. Activated NMDA can cause MAPK (mitogen-activated protein kinase) to be released. This protein has been called the master control switch of nerve-injury pain. MAPK causes more and more pain-generating chemicals such as glutamate to be produced, and at the same time increases the sensitivity of pain receptors to those very chemicals.
Long Level 2 Neuron Windup. Once MAPK is activated you need less and less glutamate to experience a given level of pain. This form of windup starts a vicious cycle of less glutamate and more pain that causes long Level 2 neurons to fire out of control, creating overwhelming pain.
Because Stage 2 is where really serious pain begins, many visualization statements are generated here. They address ion channels, interneuron windup, NMDA and MAPK activation, and long level 2 neuron windup, possibly helping to relieve pain.
Stage 3 (Thalamus to Cortex): Driving the Pain Home
If the signal leaves the Level 2 neurons with NMDA activated and long Level 2 neurons in windup, a strong pain signal is transmitted to your Level 3 nerve cells. If ion channels exist in Level 3 neurons, the signal can be made even more intense.
In Stage 3, the signal that arrived at the thalamus from Stage 2 is processed and then forwarded on to the cerebral cortex. Once the signal reaches the cortex you experience the full force of the pain.
Visualizations at this stage that address ion channels and the thalamus may help to ease pain.
Limiting the Transmission of Pain Emotion Signals
The emotional signals associated with your pain are transmitted to your brain using different signal pathways from those described above. Like the pain channels themselves, the pain emotion conduits have leverage points where it may be possible to reduce the signal. Visualizations targeted at each of those points may possibly help to reduce the emotional suffering that goes along with chronic pain.
Engaging the Subconscious
The subconscious is quite powerful. When programmed through the very visualizations that it suggests, it may be able to turn episodes of uncontrolled pain into events over which you have a degree of control.
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